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Concurrently, loss of Treg-mediated suppression permits Th17 activation and IL-17 release, driving robust neutrophil recruitment and destructive inflammation that promotes a more severe and refractory disease phenotype

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Sites of action of interleukins (IL-7, IL-3, and IL-5), stem cell factor (SCF), thrombopoietin (TPO), FLT-3 ligand (a growth factor), granulocyte-macrophage colony-stimulating factor (GM-CSF), erythropoietin (EPO), monocyte colony-stimulating factor (M-CSF), and granulocyte colony-stimulating factor (G-CSF) are shown